Liver and its Functions - Liver Conditions -Liver Cirrhosis

The main function of the liver is that the body should pass through the digestive path before passing the rest of the body

  • Manufacturers provide blood protein that helps in functioning of cloning, oxygen transport, and immune system.
  • Store additional nutrients and some of the blood nutrients are returned.
  • Manufacturers of bone, have a substance to help digestive food.
  • Helping body shop sugar (glucose) in glucose form.
  • Recognizing the body of bleeding in blood, including drugs and alcohol.
  • Reducing shy fat and generating cholesterol.


  • Hepatitis: Liver infections, usually due to hepatitis A, B, and C hepatitis, include non-profit causes including heavy drinking, drug, allergic reactions or obesity.
  • Sarrassas: For any reason, liver can lead to long term damage, which is called surgeon. The liver then failed to work properly.
  • Lever cancer: The liver cancer, the most common type of hepatopular carcomoma, is almost always after rotation is present.
  • Liver failure: Lever failure has many factors including infections, genetic diseases, and optimal wines.
  • Oscar: As a result of Syrusha, the liver jiggers (ascites) enter the stomach, which cause many and heavy.
  • Galeston: If a liver sweep sticks in a jaw, then hepatitis and thin duct infections can be the result.
  • Hemocomatosis: Hemocomatitis allows iron to collect liver, damages it. The iron also stores the body, which produces multiple health problems.
  • Early circular cholangitis: An unusual disease with unknown events, the basic sclerosing cholangitis causes inflammation and scarf in thin dyes in the liver.
  • Primary billyceric surros: In this unusual disorder, a clear procedure gradually destroys the balls of liver in the liver. Permanent liver scarf (soursis) is finally ready.


Survorus is constantly being liver due to the loss of long-term liver. The score tissue replaces the healthy tissue in the liver and the liver works properly.


  • Wine consumption and some drugs.
  • Fatty liver obesity and diabetes.
  • Liver's chronic viral infections (type of hepatitis B, C, and D; hepatitis D is extremely rare).
  • Non-alcoholic Steatohepatitis (NASH).
  • Balari barrier (Sustainable Rebrities, Blurry Orthur).
  • Other reasons
  • Automammone liver disease - Automotive hepatitis, primary blood surgery or basic sliding cololants (PSC).
  • Some rarely genetic conditions - such as hemochromatosis and ovoline disease, lack of Alpha 1 Anti Tititis.
  • Due to any condition that due to the prevention of ceiling trains - for example, thaw detectors or cancer cancer.
  • Hungry Chorei Syndrome - Due to preventing blood from liver prevention from blood.


  • The sign of the pathway of the Sarrasses marks the development of scan tissue that replaces the common parenchyme.
  • This score prevents tissue blood portal flow due to which the organism usually tries, and is the initial form of the phosphorus syrus.
  • There are several types of cells, set-ups and mrnNAs in the introduction and development of liver fiberus and syrusus.


  • HSBC, which is commonly known as fat-storage cells, i-cells, lipocytes, pressioselal cells, or vitamin A-rich cells, remains in place of common liver and is the main function. Vitamin A and other retinoids are storage.
  • Hepatile Cell Cell Cell (HSC) activity is an important event in Fabrosis. The liver circuscular endothelial cells of the defenestration and capillarization are important auxiliary factors for hepatitis in liver surgery.
  • Recent research shows the role of Steel Cell, a cell type that usually develops the variant A, in the development of syrup.
  • Damage to hepatate pneumonia (due to inflammation) leads to activating the steel cell, which prevents blood flow through the production of phosphorus (through the production of myofibrblasts) and circulation.


  • KCs are also known as Bourkors, Kosovo cells and rich macroscopes, the part of the reticuloendothelial system (RES) which is specialized in macrophages located in the lining walls of the liver of sinusoids.
  • Chemistry can be activated by many injured elements such as viral infections, alcohol, high-fat diet, and iron detection.
  • Activate the Kuper cells of the cells, destroy the hepatitis and promote HSC activities and promote the formation of phosphorus.


  • Repeat reproductive cycles of appipotts and mutatets take part in parrorsensus of cytosis.
  • To continue harmful hepatitis reactions oxygen species (ROS) and fertilizer arbitrators, promote HSC activity and promote the verbal process of your audio bubbles.
  • Apparatus of hepatocytes is a common event in liver injury and it forms part of tissues.


  • Liver Sarcosis H.Sc and Fibrengensis's organization is arcetized by a complex network of Sacocano Interested Signaling Paths.
  • PDGF is a strong museum for all polyptitidal growth factors. Four members of the PDGF family, PDG FA, -B, -C and -D. PDG F and its receptors are significantly affected by the fungus tissues, and their activity increases with the degree of liver rabies.
  • TGF-β (Basically TGF-β1) is the most popular known for hebacco reberysis. TGF-β is mainly synthesized by HCPC / Myofibroblasts, KCs, LSECs, and hepatitis in liver.
  • TNF-α is mainly developed by Manuki, Macrograde, HSC, and KCs. Their cells have prophthactic activities and the effects of svetokicus. HSCs and ECM plays an important role in the recycling activities
  • Interface and LL are included
  • Recently, the MHRN has got a major role in Frossover and Survey as a transmission regulator.

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